CDC logo
Safer Healthier People
CDC Home CDC Search CDC Health Topics A-Z
NIOSH - National Institute for Occupational Safety and Health

Skip navigation links Search NIOSH  |  NIOSH Home  |  NIOSH Topics  |  Site Index  |  Databases and Information Resources  |  NIOSH Products  |  Contact Us

Search for NIOSH Publications: NIOSHTIC-2

NIOSHTIC-2 Search Results

      Advanced Search  |  Help  |  About  |  Feedback 
Terms: manganese
16 - 16 of 490 Bibliographic entries
Save All   |   Save Page
View Saved    |    Download
Select check boxes to automatically save entries, or use 'save all' or 'save page' links above.
Back to Results
Association of genetic variations in antioxidant enzyme genes with diisocyanate-induced asthma in exposed workers.
Authors
Yucesoy-B; Johnson-VJ; Fluharty-K; Slaven-J; Lummus-ZL; Kissling-GE; Germolec-DR; Luster-MI; Bernstein-DI
Source
Toxicologist 2011 Mar; 120(Suppl 2):293
Link
http://www.toxicology.org/ms/AMpubs.asp 
NIOSHTIC No.
20038513 
Abstract
Diisocyanates are the most common cause of occupational asthma induced by low molecular- weight agents. It has been shown that diisocyanates induce oxidative stress when they react with specific proteins in human airway epithelial cells and this process contributes to the pathogenesis of diisocyanate-induced asthma (DA). In this respect, variability in the antioxidant defenses may play a role in DA susceptibility. A case-control study was conducted to investigate whether genetic variations within antioxidant enzyme genes, glutathione S-transferases (GSTM1- GSTT1, GSTM3, GSTP1), manganese superoxide dismutase (MnSOD) and microsomal epoxide hydrolase (EPHX1), play a role in susceptibility to DA. The study population consisted of 252 workers exposed to diisocyanates (hexamethylene diisocyanate, methylene diphenyl diisocyanate, and toluene diisocyanate) of which 102 were diagnosed with DA based on a positive specific inhalation challenge. Genotype analysis was performed on genomic DNA, using a 5’ nuclease PCR assay. The EPHX1 SNP (rs2740171) and GSTT1 gene deletion were associated with altered risk of developing DA after adjusting for potential confounders. Since EPHX1 and GSTT1 genes are important components of lung defense against oxidative stress, variations in these genes which regulate their expression may represent important disease modifiers and contribute to DA susceptibility.
Keywords
Allergic-reactions; Allergies; Biological-effects; Genetic-factors; Inhalation-studies; Lung-function; Lung-irritants; Molecular-biology; Molecular-structure; Oxidative-metabolism; Oxidative-processes; Oxidative-processes; Physiological-effects; Pulmonary-function; Pulmonary-system; Quantitative-analysis; Respiratory-hypersensitivity; Respiratory-irritants; Risk-analysis; Risk-factors; Statistical-analysis; Tissue-disorders
Publication Date
20110301
Document Type
Abstract
Funding Type
Grant
Fiscal Year
2011
NTIS Accession No.
NTIS Price
Identifying No.
Grant-Number-R01-OH-008795
ISSN
1096-6080
NIOSH Division
HELD
Priority Area
Healthcare and Social Assistance; Services
Source Name
The Toxicologist. Society of Toxicology 50th Annual Meeting and ToxExpo, Washington, DC, March 6-10, 2011
State
DC; OH; WV
Performing Organization
University of Cincinnati
Page 16 of 490

File Formats Help:

Adobe PDF file
How do I view different file formats (PDF, DOC, PPT, MPEG) on this site?double arrows.