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 3

Gene expression profiling and pathway analyses reveal molecular signatures and relationships underlying enhanced methamphetamine neurotoxicity caused by protracted corticosterone exposure.

Authors

Kelly-KA; Miller-DB; O'Callaghan-JP

Source

Neuroscience 2012: 42nd Annual Meeting of the Society for Neuroscience, October 13-17, 2012, New Orleans, Louisiana. Washington, DC: Society for Neuroscience, 2012 Oct; :63.16/Q10

 4

Chronic exposure to corticosterone enhances the neuroinflammatory and neurotoxic responses to methamphetamine.

Authors

Kelly-KA; Miller-DB; Bowyer-JF; O'Callaghan-JP

Source

J Neurochem 2012 Sep; 122(5):995-1009

 5

In vivo stress and chronic glucocorticoid exposure influence the neuroinflammation and dopaminergic neurotoxicity associated with methamphetamine.

Authors

Miller-DB; Kelly-KA; Bowyer-JF; O'Callaghan-JP

Source

Toxicologist 2012 Mar; 126(Suppl 1):220-221

 6

Chronic exposure to glucocorticoids primes the CNS proinflammatory response in methamphetamine neurotoxicity.

Authors

Kelly-KA; Miller-DB; James-OP

Source

Toxicologist 2011 Mar; 120(Suppl 2):37-38

 7

Chronic exposure to CORT primes the CNS proinflammatory response in MPTP and METH models of neurotoxicity.

Authors

O'Callaghan-JP; Miller-DB

Source

Toxicologist 2010 Mar; 114(1):271

 8

Decreased neuronal damage and glial reactivity in an animal model of stress.

Authors

Benkovic-SA; O'Callaghan-JP; Miller-DB

Source

Toxicologist 2010 Mar; 114(1):126

 9

Protracted exposure to supraphysiological levels of corticosterone does not cause neuronal loss or damage and protects against kainic acid-induced neurotoxicity in the hippocampus of C57BL/6J mice.

Authors

Benkovic-A; O'Callaghan-JP; Miller-DB

Source

Neurotoxicology 2009 Nov; 30(6):965-976

 10

High physiological levels of the stress hormone, corticosterone (CORT), exacerbate brain inflammatory responses to lipopolysaccharide (LPS) and to the nerve agent, DFP: role in Gulf War Illness?

Authors

O'Callaghan-JP; Miller-DB; Lasley-SM

Source

Toxicologist 2009 Mar; 108(1):13

 11

Corticosterone attenuates hippocampal neurotoxicity and reactive gliosis through regulation of the blood-brain barrier in C57BL/6J mice treated with kainic acid.

Authors

Benkovic-SA; O'Callaghan-JP; Miller-DB

Source

Toxicologist 2009 Mar; 108(1):449